EPO and triple-A syndrome: These results have several important implications: first, β2AR agonism with formoterol had no effect on the morphology of mouse aorta; second, the beneficial effects of medium‐dose formoterol on AAA came into play only in the presence of EPO stimulation; and third, the null effect of high‐dose formoterol on AAA development cannot be explained by the direct harmful effect of high‐dose formoterol on mouse aortic tissues.