DPYSL2 and Parkinson disease: Using a mouse model of PD, Kentaro Togashi et al. explored the impact of phosphorylation of Collapsin Response Mediator Protein 2 (CRMP2) on PD pathogenesis and found that genetically inhibiting CRMP2 phosphorylation ameliorated the condition of mice exposed to a PD-inducing neurotoxin [44].