MYB and metabolic dysfunction-associated steatotic liver disease: Moreover, in the Q1 cluster, the enriched cellular component (CC) annotations included mitochondria, nucleolus and cell body; the enriched KEGG pathways included nonalcoholic fatty liver disease, nucleocytoplasmic transport and MAPK signaling pathway; and the enriched protein domains included Myb-like DNA binding domain and C2H2-type zinc finger (Fig. S7E-G).