SLC7A11 and lung cancer: RBMS1 has been reported to regulate DNA replication, DNA transcription, and RNA stability.[16, 17, 18] Recently, we revealed that RBMS1 could modulate the translation of SLC7A11 by binding to its 3′‐UTR.[15] When RBMS1 is depleted, the translation of SLC7A11 was inhibited, and the reduced SLC7A11 promoted ferroptosis, thereby inhibiting lung cancer proliferation in cultured cancer cells, xenograft mice, and genetically conditional knockout mice.[15] However, the function of RBMS1 in NSCLC metastasis is less understood.