Furthermore, miR-30a up-regulation reduces AF-induced myocardial fibrosis by targeting snail family transcriptional repressor 1 (SNAIL1) in a rabbit model of experimental AF [102], whereas miR-30c overexpression attenuates Tgf-β1-induced atrial fibrosis by targeting transforming growth factor beta receptor 2 (TgfβrII) [103]. Here, TGFB1 is linked to atrial fibrillation.