The systemic response to the infection in our study cohort was affirmed by the high variation observed in platelet, neutrophil, lymphocyte, monocyte counts, alanine aminotransferase, aspartate aminotransferase, gamma-glutamyl transferase, bilirubin, lactate dehydrogenase, creatinine, and C-reactive protein clinical measurements, which indicate renal and hepatic dysfunction and inflammation [26,27]. Here, CRP is linked to infection.