The uncontrolled aggregations of misfolded amyloidogenic proteins, e.g., the tau in the neurons of the brain and the amylin in the beta cells of the pancreas, have separately been linked to the early molecular events of the progression of Alzheimer’s (AZ) and type-2 diabetes (T2D), respectively [1,2,3]. The gene discussed is MAPT; the disease is type 2 diabetes mellitus.