Moreover, a study with IL-4- and IL-13-treated macrophages stimulated with IL-33 (an asthma cellular model) induced an increase in the expression of miR-155-5p, along with an increase in inflammatory mediators (Ccl3, Ccl5, Ccl17, Ccl24, and Il1b) compared to macrophages without pretreatment [89]. The gene discussed is IL1B; the disease is asthma.