The coadministration of the miR-146a-5p mimic in a mouse asthma model promoted the decrease in AHR (increase in both eosinophil, neutrophil, macrophage, and lymphocyte infiltration and inflammatory mediators, namely OVA-specific IgE and Th2 cytokines: IL-4, IL-5, and IL-13 in BALF) by the suppression of ILC2 responses [71]. The gene discussed is IL4; the disease is asthma.