Our data supports the hypothesis that in MF cells, uniquely, higher GZ17-6.02 concentrations regulate novel mechanisms, independent of the ATM-AMPK-mTORC1/ULK1 pathway we have previously observed using GZ17-6.02 in solid tumor cells, to further enhance macroautophagy and MF tumor cell killing. Here, ULK1 is linked to neoplasm.