However, we did not detect other genetic abnormalities common in PCa and presented in the literature, such as mutations of genes involved in the repair of DNA damage (BRCA1/2, ATM, BAP1, RAD50, FANCE, PALB2), chromatin remodeling (ARID1A, PBRM1, ARID2, KMT2D, KMT2C, SMARCA4, SETD2) and cell cycle control pathways (CDKN2A, CCND1, CCNE1), as well as in Ink4a, LKB1, MLH1, PRSS1, BRAF, MAPK, PI3K, Akt, VEGF and IGF1R genes. This evidence concerns the gene KMT2D and posterior cortical atrophy.