CDH1 and non-small cell lung carcinoma: Among these, few have been known to accelerate EMT for instance, Wang et al. showed that E-cadherin is a direct target of miR-9 and its upregulation had led to a consecutive downregulation of the expression of E-cadherin in NSCLC tissues [50] Further, miR-10b have been seen promoting EMT and invasion by targeting HOXD10, a transcription factor that acts as a suppressor of EMT [51].