The asymmetric dimethylarginine produced by PRMT1 inhibits the inducible nitric oxide synthase, reducing the production of nitrogen oxides and sulfur oxides, thereby weakening the oxidative stress and inflammatory response of the liver under the influence of alcohol, which in turn reduces cell death, inflammation, and the Wnt/β-Catenin signaling pathway activation, as well as tumor growth [118]. The gene discussed is PRMT1; the disease is neoplasm.