Ectopic FUT4 expression reduced the number of N-cadherin:β-catenin and N-cadherin:δ1-catenin interactions per melanoma cell, whereas the depletion of FUT4 or treatment with 2F-peracetyl-fucose (2FF), a pharmacological inhibitor of FUTs39, enhanced these junction complexes (Fig. 3d, e and Supplementary Fig. 3e–h). The gene discussed is FUT4; the disease is melanoma.