Based on previous findings, VIM is reported to be expressed 11‐fold greater in glioblastoma than in non‐tumor brain tissue.[34] In addition, A2M and CA9 have also been found to be up‐regulated in glioblastoma tissue.[35, 36] Before running the full panel, we validated each antibody with primary and secondary antibody staining (Figure S2, Supporting Information). The gene discussed is CA9; the disease is neoplasm.