In both SIDS infants and controls, we examined the prevalence of low 5-HT2A/C and 5-HT1A binding within key subnetworks vital for blood pressure regulation (NTS-MAO-GC; that is “olivocerebellar” subnetwork) as well as the subnetwork containing the 5-HT source neurons (ROb/RMg-GC-PGCL-IRZ subnetwork) (53). This evidence concerns the gene HTR2A and sudden infant death syndrome.