NFKB1 and nasopharyngeal carcinoma: Similarly, another study highlighted that, in the nasopharynx carcinoma model, the EVs could package latent membrane protein 1 (LMP1) to transform normal fibroblasts into CAFs by virtue of the NF-κB P65 signaling cascade, and the activated CAFs were essential for promoting the proliferation and migration of nasopharyngeal carcinoma cells via aerobic glycolysis and autophagy.