It is reported that overexpression of BACH1 stimulated by long non-coding RNAs (lncRNA) small nucleolar RNA host gene 5 (SNHG5) regulates the levels of proteins related to glucose metabolism by targeting miR-299, thus regulating glucose consumption and lactate secretion, thereby facilitating the growth and glycolysis of breast cancer cells [66]. The gene discussed is BACH1; the disease is breast carcinoma.