Then, the GO analysis suggested that genes correlated with SEMA3C were enriched in numerous cancer-related biofunctions such as cell adhesion, positive regulation of JAK-STAT, positive regulation of stem cell maintenance, and positive regulation of NF-κB activity, indicating that SEMA3C played a role in oncogenesis through these pathways (Fig. 6a–c). This evidence concerns the gene SOAT1 and cancer.