These modules were expressed in both 170-infected animals at predominantly 4 weeks post-infection and contained genes implicated in DC trafficking to secondary lymphoid tissues, including CCR7 and IL7R [58], pro-inflammatory chemokine CXCL16, and molecules involved in communication with T cells, including CD101 and CCL22 [59, 60] (Fig. 1G). This evidence concerns the gene CXCL16 and infection.