Given that LL-37 is involved in pathogenesis of lupus and rheumatoid arthritis, the notion that SARS-CoV-2 peptide fragments can imitate LL-37 may be conceptually salient for understanding why the immune systems of COVID-19 patients resemble those of people with autoimmune disorders like lupus and rheumatoid arthritis (45). This evidence concerns the gene CAMP and COVID-19.