This has been shown to have an antitumor metastasis effect in BRCA.361 Overexpression of DHX38 can promote the normal splicing of RELL2 pre-mRNA and the synthesis of RELL2 protein, thereby inhibiting tumor progression.362 However, this study did not explore the specific mechanism, indicating a need for further research to explore the therapeutic potential of this target for PDAC. Here, RELL2 is linked to neoplasm.