INSR and glioblastoma: HNRNPA2B1, for instance, promotes several exons exclusion, including recepteur d’Origine Nantais (RON) exon 11, insulin receptor (INSR) exon 11, CASP8 and FADD-like apoptosis regulator (CFLAR; C-FLIP) exon 7, caspase 9 (CASP9) exons 3–6 as well as WW domain-containing oxidoreductase (WWOX) exons 6–8.199 Inhibition of HNRNPA2B1200,201 reduces GBM cell vitality, adhesion, migration, and invasion.