BRAF and neoplasm: Studies have shown increased activation of BRAF–KIAA fusion, in the cerebellar and optic pathways in tumour progression in paediatric patients91 and functional inactivation of MAPK/ERK components, facilitates the neuronal progression of astrocytic molecular markers such as SOX10, platelet‐derived growth factor (PDGFα) receptors and neural/glial antigen 2 (NG2) proteoglycans.92