ACE and renal tubular acidosis: In the latter group, the renal tubular excretion of potassium can be compromised through disruption of RAAS, e.g., medications such as angiotensin-converting enzyme inhibitors (ACE inhibitors), or tubular resistance to the action of aldosterone (e.g. renal tubular acidosis), leading to altered potassium excretion and subsequently hyperkalemia [2, 13].