Equally, in Burkitt lymphoma, mantle cell lymphoma, and diffuse large B-cell lymphoma, where the mechanisms of chromosomal translocation are similar [ie, resulting from errors in V(D)J recombination],31 overexpression of tumor-specific oncogenes MYC (MYC proto-oncogene), CCND1 (cyclin D1), and BCL6 (BCL6 transcription repressor) could also be targeted using this enrichment strategy. This evidence concerns the gene MYC and diffuse large B-cell lymphoma.