FGF14 and late-onset spinocerebellar ataxia 27b: Since RNA and protein expression of FGF14 was found to be decreased in post‐mortem cerebellum samples and induced pluripotent stem cell‐derived motor neurons of SCA27B patients,21 the current evidence suggests that SCA27B is caused by loss of function of FGF14, potentially through inhibition of transcription by the sticky DNA secondary structure.