FGF14 and late-onset spinocerebellar ataxia 27b: The relatively elevated population allele frequency of FGF14 repeat expansions21, 22, 32 increases the likelihood that a co‐morbid neurodegenerative disease or a second pathogenic variant in another ataxia gene co‐occur in a single patient, thus complexifying the underlying phenotype (which should not be interpreted as being indicative of a broader phenotypic spectrum of SCA27B).30