DNM2 and early-onset autosomal dominant Alzheimer disease: It has been shown that transient hypoxia is associated with hyperphosphorylation of cytoskeletal proteins and abnormalities in the functioning of neuronal mitochondria, which may lead to Alzheimer’s disease-like changes and may also cause an increase in the level of amyloid, which is an important element in the pathology of Alzheimer’s disease [115].