As expected, the key Wnt-effector β-catenin and two kinases, GSK3β and CK1α, were both incorporated into the condensates in CRC cells (Fig. 3A–C and S6A), and truncated APC proteins could also bind β-catenin to form condensates (Figs. 2E and 3D). The gene discussed is GSK3B; the disease is colorectal carcinoma.