NTRK2 and pneumocystosis: Although the influence of glutamatergic antipsychotics on BDNF release remains to be elucidated, we show in the present study a key role of TrkB activation in the ability of glutamatergic antipsychotics to alleviate cognitive and negative symptoms in mice treated subchronically with PCP, providing a first link between BDNF-TrkB signaling and therapeutic response to antipsychotics.