HIF1A and hematoma: Based on the lack of blood vessels, it was typically assumed that these areas are hypoxic,[44, 77] yet in contradiction with this assumption, hypoxia is known to promote the expression of hypoxia‐inducible factor‐1α (HIF‐1α), and thus angiogenesis.[78‐–82] As the cells entrapped in the blood clot[10, 83] already require a substantial supply of nutrients to survive, and play a major role in the hematoma remodeling, we suggest an alternative to explain the delayed hematoma vascularization under semi‐rigid fixation.