We observed at P21 that loss of Foxj1 in constitutive knockout mice resulted in a severe disfigurement of the olfactory system at the gross anatomical level; largely undeveloped turbinates were formed, and the entire nasal cavity was filled with DAPI-positive cellular masses composed of neutrophils (S3A, S3A’ and S3D Fig), similarly to primary ciliary dyskinesia patients [50]. This evidence concerns the gene FOXJ1 and primary ciliary dyskinesia.