Other studies have observed that natural cytotoxicity receptors (e.g., NKp30, NKp44, and NKp46), NK‐activating receptors (e.g., sDNAM‐1 and NKG2D), and activating killer cell immunoglobulin‐like receptors were bound by the overexpression ligands of cancer cells and that NK cells were activated and produced many cytotoxic granules and pro‐inflammatory factors for lysing cancer cells.11 The gene discussed is NCR3; the disease is cancer.