BRAF and gastric cancer: Similarly, our research group revealed that MAGOH was highly expressed in GC cells, that its knockdown inhibited the occurrence of GC by mediating b-RAF/MEK/ERK signaling and that the double knockdown of MAGOH and MAGOHB exerted better antitumor effects than did the single knockdown of MAGOH and MAGOHB, thus providing a potential new strategy for the treatment of GC [24].