The results revealed that CXCL2, CXCL3, CCL20, CCL25 and CCL28, which played key role in recruiting T lymphocytes to tumor microenvironment (Tosti et al. 2020; Chen, et al. 2020; Gong et al. 2019), were negatively correlated with LOXL1 expression in in dependent cohorts (Fig. 5G-I). The gene discussed is CXCL2; the disease is neoplasm.