In addition, other inflammation‐related pathways (e.g. inflammatory response, interferon‐α response, interferon‐γ response and IL2‐STAT5 signalling) were significantly upregulated in some cell subsets of the polyp subtypes, whereas oxidative phosphorylation and MYC targets V1 were significantly downregulated in most cell subsets of all three polyp subtypes. The gene discussed is IL2; the disease is polyp.