To further determine whether APOL1 is a direct and functional mediator of the HIF2α /LINC02609 axis promoting tumor progression and lipid storage, we performed a rescue experiment by co-transfecting with HIF2α shRNA (versus the negative control), LINC02609 shRNA (versus the negative control) and APOL1 (versus the negative control) into 786-O and A498 cells. This evidence concerns the gene APOL1 and neoplasm.