In 2010, these allowed the classification of glioblastomas into subtypes according to their genetic expression profiles: classical (EGFR overexpression, CDKN2A deletion, and a lack of TP53 mutations), mesenchymal (altered NF1, PTEN mutations, a high activity of MET and CD44, and MERTK genes), proneural (altered PDGFRA, mutated TP53, point mutations in IDH1 and the increased expression of OLIG2), and neural (expression of neural markers such as NEFL, GABRA1, SYT1, and SLC12A5) [5]. Here, EGFR is linked to glioblastoma.