Veltman et al. reported that elevated oxidative stress, an abnormal lipid fingerprint, and enhanced pro-inflammatory signaling in well-differentiated bronchial epithelial cells isolated from neonatal CFTR KO pigs and adult CF patients undergoing lung transplantation were partially corrected after incubation with a combination of ETI and antioxidants [18]. This evidence concerns the gene CFTR and cystic fibrosis.