Rhabdomyolysis is a clinical syndrome that comprises the destruction of skeletal muscle with outflow of intracellular muscle content into the bloodstream [1] and a complex condition characterized by the breakdown of damaged muscle tissues, resulting in the release of intracellular muscle contents, including myoglobin, creatine phosphokinase (CPK), lactate dehydrogenase, and electrolytes [2,3]. This evidence concerns the gene PIK3C2A and rhabdomyolysis.