AD patients had significantly higher abundances of thymosin β4, α-defensins—1, 2, 3, and sum of α-defensins, histatin 1 mono- and non-phosphorylated, statherin 2P, des 1-9 and des 1-13, P-C peptide, cystatin A, B-SSG, total cystatin B monomer, cystatin B S-S dimer, total cystatin B, S100A8-SNO, sum of S100A8-A8SNO, S100A9s, sum of S100A9s, and total S100A9 (s + l) compared to controls, and there were analogically significantly lower abundances of PRP1 0P. The gene discussed is CSTA; the disease is Alzheimer disease.