A variety of lncRNAs, including growth arrest-specific transcript 5 (GAS5), nuclear paraspeckle assembly transcript 1 (NEAT1), and metastasis-associated lung adenocarcinoma transcript 1 (MALAT1), are implicated in the pathogenesis of SLE [137,138,139]. This evidence concerns the gene MALAT1 and systemic lupus erythematosus.