IL1B and infection: eHSP-27 modulates the expression of the IκB (inhibitor of nuclear factor kappa B) with subsequent reduction of inflammation, oxidative stress, and apoptosis; on the contrary, during the infection, eHSP-60 and eHSP-70 promote NF-κβ expression with subsequent increased release of IL-1β, TNFα, and other pro-inflammatory molecules [37].