The IL-17 signaling pathway plays an essential role in tumor progression, exerting its influence on tumor cells through multiple avenues; for instance, IL-17A, which is predominantly synthesized by immune cells, particularly Th17 cells, has been linked to HGSOC carcinogenesis by upregulating MTA1 and enhancing the self-renewal capacity of CD133-positive HGSOC stem-like cells [41,42,43]. This evidence concerns the gene PROM1 and neoplasm.