It is known that the inhibitors of proteins involved in signal transduction by RAS-RAF-MEK-ERK and PI3K-AKT/PKB-mTOR signaling pathways significantly increases the sensitivity of glioma cells to the induction of apoptosis [19,20] Therefore, in addition to a single application of furanocoumarins, they were combined with the RAF kinase inhibitor—sorafenib and the PI3K kinase inhibitor—LY294002 and the microscopic observations of characteristic morphological changes were conducted (Figure 4). The gene discussed is AKT1; the disease is central nervous system cancer.