Survival curves showed that patients with low levels of these cellular markers and high levels of TGFB2 mRNA exhibited worse survival rates, which suggests that cellular-level responses can be targeted by IFN-γ; these responses have the potential to enhance the anti-tumor response when immunosuppression is lifted owing to elevated TGFB2 levels [36]. The gene discussed is TGFB2; the disease is neoplasm.