To further filter the dysregulated pathways that are important in the development of DCM, we used publicly available gene expression data in human DCM and normal heart samples and identified differentially expressed genes followed by analysis of their enriched pathways and processes, which revealed 15 common pathways that were significantly enriched by Rpl3l-correlated genes in mouse and differential genes in human heart (Figure 4). The gene discussed is RPL3L; the disease is familial dilated cardiomyopathy.