CSF1R and breast carcinoma: A partial epithelial-to-mesenchymal transition (EMT) characterized by the expression of both Vimentin and E-cadherin was observed in inflammatory breast cancer, where treatment with the CSF-1R inhibitor BLZ945 reduced the spindle-like phenotype of cancer cells and reversed the partial EMT, indicating the role of CSF-1R in breast cancer invasiveness [59].