For example, Hwang et al. found that ΔdblGATA mice were resistant to experimental autoimmune encephalomyelitis (EAE), a murine model of multiple sclerosis, but they reported that the impaired antigen-presenting cell function but not the deficiency of eosinophils resulted in the resistance to EAE due to GATA-1’s roles on monocyte lineages [36]. The gene discussed is GATA1; the disease is experimental autoimmune encephalomyelitis.