Viral infection-induced FUT8 expression was dependent on EGFR, AKT, and SNAIL, as HCV-infection-induced upregulation of FUT8 mRNA levels (Fig. 2e) and FUT8 promoter activation (Fig. 2f) were attenuated in Huh7 cells after silencing EGFR, AKT, or SNAIL. This evidence concerns the gene SNAI1 and infection.