AKT1 and viral infectious disease: Viral infection-induced FUT8 expression was dependent on EGFR, AKT, and SNAIL, as HCV-infection-induced upregulation of FUT8 mRNA levels (Fig. 2e) and FUT8 promoter activation (Fig. 2f) were attenuated in Huh7 cells after silencing EGFR, AKT, or SNAIL.