According to Jin’s et al. research (34), baicalin reportedly prevented microglia from activating, decreased neuroinflammation, and postponed neuronal loss; inhibition of NLRP3 inflammatory vesicles and blocking of the TLR4/NF-B signaling pathway may also play a role in baicalin’s therapeutic action on AD. The gene discussed is NLRP3; the disease is Alzheimer disease.