Previous studies indicated that YTHDF2 stabilizes VEGFA transcripts in glioblastoma stem cells in an m6A‐dependent manner.[11] The m6A site at IRES‐A suppresses uORF impairment while facilitating G‐quadruplex‐induced translation of VEGFA in lung cancers.[52] In the current study, we found that the mRNA expression of VEGFA was upregulated by YTHDF2 via ETV5 in HCC. This evidence concerns the gene VEGFA and lung carcinoma.